Pneumothorax

 

  • Background

    Pathophysiology

    • A defect in the visceral pleura causes air to enter the pleural space from the lungs. The elastic recoil of the lungs then causes them to deflate/collapse.
    • A simple pneumothorax is a non-expanding collection of air. In a tension pneumothorax, the communication acts like a valve, causing continued inflation.
    • Typically unilateral, but can be bilateral.
    • Once the communication is obliterated, air is gradually reabsorbed into the lung from the pleural space.

    Causes and classification

    • Primary spontaneous: no underlying cause.
    • Secondary spontaneous (due to lung disease): asthma, COPD, TB, pneumonia, lung cancer, cystic fibrosis, ILD. Also seen in connective tissue disorders like Marfan's.
    • Traumatic: iatrogenic or accidental (e.g. rib fracture).
  • Signs and symptoms

    Symptoms:

    • Acute-onset SOB.
    • Pleuritic chest pain.
    • Sudden deterioration in asthma or COPD.
    • Can be asymptomatic.
    • Tension pneumothorax → respiratory distress.

    Signs:

    • ↓Chest expansion leading to asymmetrical expansion.
    • Hyper-resonant percussion.
    • ↓Breath sounds.
    • Trachea deviated away from affected side in severe tension pneumothorax.
  • Investigations

    • Erect CXR, but skip it in tension pneumothorax.
    • CT only if diagnostic uncertainty.
    • USS can be used in supine trauma patients.
  • Management

    Primary pneumothorax:

    • Conservative treatment if small and no SOB, as they usually self resolve. 'Small' is a margin of <2 cm on CXR between lung margin and chest wall at level of hilum.
    • Otherwise, needle aspiration, then chest drain if unsuccessful.

    Secondary pneumothorax:

    • Conservative treatment if <1 cm and no SOB.
    • Needle aspiration if 1-2 cm and no SOB. Chest drain if unsuccessful.
    • Chest drain if >2 cm or SOB.

    Tension pneumothorax:

    • 100% O2.
    • Needle aspiration for instant relief, then proceed to chest drain.

    Procedures

    Needle aspiration (aka thoracentesis):

    • Large bore cannula (16-18G, 14G if tension) into the lower 2nd intercostal space at the mid-clavicular line.
    • In simple pneumothorax, attach a tap and syringe for aspiration.
    • In tension pneumothorax, just remove the stylet for instant relief.
    • Not used in traumatic pneumothorax.

    Chest drain:

    • Inserted in the triangle of safety, in the 4th or 5th intercostal space, anterior to the mid-axillary line.
    • First infiltrate lidocaine 1% all the way through to the pleural space.
    • Small-bore drain via Seldinger technique is first choice. Large-bore drain via blunt dissection is only for very large air leaks.
    • Unlike for pleural effusions, ultrasound-guidance is not a requirement.
  • Atelectasis and lung collapse

    Atelectasis, literally incomplete (ateles) expansion (ektasis), refers to collapse or incomplete expansion of the lung parenchyma.

    Passive (aka relaxation) atelectasis

    • Loss of contact between the visceral and parietal pleurae.
    • Causes: pneumothorax, pleural effusion.
    • Can lead to the collapse of an entire lung.

    Obstructive atelectasis

    Collapse secondary to the occlusion of a bronchus, as the air beyond the obstruction is gradually absorbed and the airways collapse.

    Causes:

    • Tumour
    • Aspirated foreign body, commonly right main bronchus.
    • Mucous plug e.g. in asthma.
    • Post-operative retention of secretions.
    • Tracheobronchial lymphadenopathy.

    Leads to lobar or segmental collapse.

    Linear atelectasis

    Small amount of incomplete expansion resulting from restricted breathing ('respiratory splinting').

    Causes:

    • Post-op pain.
    • Rib fracture.
    • Pleuritic chest pain.

    Compressive atelectasis

    Space occupying lesion compresses the lung.

    Causes:

    • Chest wall tumour.
    • Pleural effusion.
    • Elevated hemidiaphragm.

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